Lauricidin (8 oz)

The antiviral, antibacterial, and antiprotozoal properties of lauric acid and monolaurin have been recognized for nearly three decades. Lauricidin® is the only monolaurin that has been clinically tested.* Through advanced technology, this natural substance is now available in a pearlized 100% pure form rather than as monolaurin capsules.*
Sug. Price:$32.50
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Lauricidin® is the only monolaurin that has been clinically tested.

Through advanced technology, this natural substance is now available in a pearlized 100% pure form rather than as monolaurin capsules.

  • The antiviral, antibacterial, and antiprotozoal properties of lauric acid and monolaurin have been recognized for nearly three decades by only a small number of researchers: their work, however, has resulted in 100 or more research papers and numerous U.S. and foreign patents. Prof. Dr. Jon J. Kabara performed the original seminal research in this area of fat research. Kabara (1968) first patented certain fatty acids (FAs) and their derivatives (e.g., monoglycerides (MGs) that can have adverse effects on various microorganisms. While nontoxic and approved as a direct food additive by the FDA, monolaurin (Lauricidin®) adversely affects bacteria, yeast, fungi, protozoa, and envelope viruses.

    Technical information on Lauricidin® (monolaurin):
  • It is important to know what is a monoglyceride like monolaurin (Lauricidin® ).For visualization, a monoglyceride is composed of glycerin and one fatty acid joined as in the drawing below. In this illustration, sn represents the glycerol carbon position, n the length and R the kind of fatty acid attached to glycerol.

  • A monoglyceride (MG) lipid of which are two kinds, will have only one fatty acid attached to the sn1(3)- or the sn2- position of glycerol. The fatty acids in the sn1 and sn3 positions are similar since they occupy a position at either end of the glycerol molecule. Therefore, not all monoglycerides are the same since the fatty acid can occupy the end position (sn1(3) of glycerol or the middle sn2 position. Their structural differences need to be emphasized. It is important therefore to remember these structural sn designations since they reflect the different biological metabolism of the two monoglycerides. Where there are two fatty acids attached to glycerin, the lipid is now known as a diglyceride (DG). If three fattyacids are attached the lipid is known as a triglyceride (TG) or simply as a fat.

  • Kabara found that the properties that determine the anti-infective action of lipids are related to their structure: e.g., free fatty acids & monoglycerides. While the monoglycerides are active; diglycerides and triglycerides (fats) are inactive. Of the saturated fatty acids, lauric acid has greater antiviral activity than caprylic acid (C-8), capric acid (C-10), or myristic acid (C-14).

  • Fatty acids and monoglycerides produce their killing/inactivating effects by several mechanisms. An early postulated mechanism was the perturbing of the plasma membrane lipid bilayer. The antiviral action attributed to monolaurin is that of fluidizing the structure in the envelope of the virus, causing the disintegration of the microbial membrane. More recent studies, indicate that one antimicrobial effect in bacteria is related to monolaurin's interference with signal transduction/toxin formation (Projan et al 1994). Another antimicrobial effect in viruses is due to lauric acid's interference with virus assembly and viral maturation (Hornung et al 1994). The third mode of action may be on the immune system itself (Witcher et al, 1993).

    Antiviral Effects: Hierholzer and Kabara (1982) first reported the antiviral activity of the monoglyceride of lauric acid (monolaurin) on viruses that affect humans.. They showed virucidal effects of monolaurin on enveloped RNA and DNA viruses. This work was done at the Center for Disease Control of the U.S. Public Health Service. This study was carried out using selected virus prototypes or recognized representative strains of enveloped human viruses. All these viruses have a lipid membrane. The presence of a lipid membrane on viruses makes them especially vulnerable to lauric acid and its derivative monolaurin. These initial findings from the Center of Disease Control (CDC) have been confirmed by many other investigators.

  • Research has shown that enveloped viruses are inactivated by added fatty acids and monoglycerides in both human and bovine milk (Isaacs et al 199 1). Others (Isaacs et al 1986, 1990, 1991, 1992; Thormar et al 1987) have confirmed Kabara's original statements concerning the effectiveness of monolaurin.

  • Some of the viruses inactivated by these lipids are the measles virus, herpes simplex virus (HSV-1 and -2), herpes family members (HIV, hepatitis C, vesicular, stomatitis virus (VSV), visna virus, and cytomegalovirus (CMV). Many of the pathogenic organisms reported to be inactivated by these antimicrobial lipids are those know to be responsible for opportunistic infections in HIV -positive individuals. For example, concurrent infection with cytomegalovirus is recognized as a serious complication for HIV positive individuals (Macallan et al 1993).

  • Thus, it would appear imperative to investigate the practical aspects and the potential benefit of a nutritional supplement such as monolaurin (Lauricidin®) for microbial infected individuals. Until now few nutritionists in mainstream nutrition community seem to have recognized the added benefit of antimicrobial lipids in the support of infected patients. These antimicrobial fatty acids and their derivatives are essentially nontoxic to man. According to the published research, lauric acid is one of the best "inactivating" fatty acids, and its monoglyceride is even more effective than the fatty acid alone (Kabara 1978, Sands et al 1978, Fletcher et al 1985, Kabara 1985).

  • It should be emphasized that lauric acid cannot be taken orally because it is severally irritating. Lauricidin® on the other hand, a derivative of lauric acid chemically bonded to glycerin to form monolaurin, can be taken orally without any problem.

    Antibacterial Effects: The potentially pathogenic bacteria inactivated by monolaurin include Listeria monocytogenes, Staphylococcus aureus, Streptococcus agalactiae, Groups A, streptococci-gram-positive organisms, and some gram-negative organisms (Vibrio parahaemolyticus and Helicobacter pylori). Decreased growth of Staphylococcus aureus and decreased production of toxic shock syndrome toxin-l was shown with monolaurin (Holland et al 1994). Monolaurin was 5000 times more inhibitory against Listeria monocytogenes than ethanol (Oh & Marshall 1993). In vitro monolaurin rapidly inactivate Helicobacter pylori. Of greater significance there appears to be very little development of resistance of the organism to the bactericidal effects (Petschow et al 1996) of these natural antimicrobials.

  • A number of fungi, yeast, and protozoa are also inactivated or killed by monolaurin. The fungi include several species of ringworm (Isaacs et al 1991). The yeast reported to be affected is Candida albicans (Isaacs et al 1991). The protozoan parasite Giardia lamblia is killed by monoglycerides from hydrolyzed human milk (Hemell et al 1986, Reiner et al 1986, Crouch et al 1991, Isaacs et al 1991). Chlamydia trachomatis is inactivated by monolaurin (Bergsson et al 1998). Hydrogels containing monocaprin/monolaurin are potent in vitro inactivators of sexually transmitted viruses such as HSV-2 and HIV-1 and bacteria such as Neisserian gonorrhea (Thormar 1999).

  • Monolaurin does not appear to have an adverse effect on desirable gut bacteria, but rather on only potentially pathogenic microorganisms. For example, Isaacs et al (1991) reported no inactivation of the common Esherichiacoli or Salmonella enteritidis by monolaurin, but major inactivation of Hemophilus influenza, Staphylococcus epidermis and Group B gram positive streptococcus.

    The Problem of Antibiotics: The phenomenal rate of prescriptions dispensed for antibiotic use, and to a lesser extent, antiviral has grown exponentially in the past several decades. Antibiotic has limited specificity and generally does not recognize “good” bacteria (often referred to as probiotics or for life) from “bad” bacteria (meaning those bacteria that may cause disease.) Antibiotics try to destroy all bacteria and are usually unsuccessful.

  • More antibiotic therapy may start perpetuating a chronic illness. The cycle of antibiotic therapy may go on for months and months, and repetitious indiscriminate use of antibiotics destroys weak bacteria and sets up the stage for the more virulent bacteria to survive (as in survival of the fittest). The new, stronger, pathogenic bacteria are now “resistant” to the established antibiotic and another antibiotic must be found to fight the new pathogen. We are rapidly approaching that point in history of having super bacteria: disease causing bacteria that are unaffected by any antibiotic. In its failure, antibiotic therapy has taken with it the health of those same individuals it strives to help.

  • The great advantage of Lauricidin® is that it does not produce resistant microorganisms during use. Not only does Lauricidin® not produce resistance but also it is known to help resistant organisms from forming.*

    Microorganisms Inactivated Under Laboratory Conditions:
  • Viruses: HIV or HIV-1, -6 Visna virus Herpes simplex virus-i (HSV-1 &2) Vesicular stomatitis virus (VSV) Measles virus Rubella virus Epstein-Barr virus (EBV) Respiratory syncytial virus Influenza virus Dengue virus (Type 1-4) Leukemia virus Cytomegalovirus (CMV) Semliki forest virus Lymphocytic choriomeningitis Human papilloma virus (HPV) Pneumovirus

  • Bacteria: Gram-positive organisms Gram-negative organisms Bacillas anthracis (Anthrax) Chlamydia pneumonia Listeria monocytogenes Neisseria gonorrhoeae Staphylococcus aureus Helicobacter pylorus Groups A, B, F & G streptococci Mycoplasma pneumonia Streptococcus agalactiae Vibrio parahaemolyticus Mycobacteria Clostridium perfringens

  • Yeasts. Fungi and Molds: Aspergillus Niger Malassezia, species Saccharomyces cerevisiae Penicillium citrinum Ringworm or tinea (Trichophyton) Candida utilis

  • A number of protozoa like Giardia lamblia are also inactivated or killed by Lauricidin®*

    DO:
  • Take Lauricidin® in small amounts until you reach your level for optimal health and self healing. This could mean from a few pellets to 1-3 blue scoops/day.
  • When air or ship traveling, you may need to increase your maintenance level.
  • When faced with stress or "coming down with something," increase the level during these times.
  • Take daily for optimal health and self healing much like vitamins.

    DON'T:
  • Do not chew the pellets.
  • Do not take too much, too soon in order to avoid a Herxheimer reaction.(die off reaction)

    Other Information:
  • Lauricidin® is a concentrated mini-pellet (~30 mg) of sn1 (3)-monolaurin. Monolaurin as a class of direct food additives is consider GRAS (Generally Regarded As Safe) by the USA FDA. Lauricidin® is nontoxic and does not interact with most drugs or supplements.

  • Because Lauricidin® is bioactive the exact amount that you need for optimal health depends on your diet and genetic make up.

  • This new field of nutrigenomics (interaction between nutrition and the human genome) will better help us understand how nutrients are able to prevent and even treat disease through targeted nutrition.

    Children (3-10 year olds): Do not chew pellets. To be taken with meals. The mini-pellets can be placed in the mouth and swallowed with water or juice. Do not take with hot liquids. For difficult situations try placing the pellets (whole or powdered) into applesauce, pudding, peanut butter, etc. For very young children it is best to start with 1-3 pellets/day for a several days before gradually increasing the dose.The usual initial level of Lauricidin® is increased gradually to twice/thrice daily for a week or two. The level can be further increased thereafter.

    The maintenance level will depend on effects seen and adjusted accordingly. Contact Dr. Kabara with any questions/comments

    Adults: Take with or after meals. The mini-pellets can be placed in the mouth and swallowed with water or juice. Do not take with hot liquids. Do not chew or take as a powder. The recommended initial level of Lauricidin® is ~0.75 gram (1/4 blue scoop) or less two or three times daily for a week before increasing the amount.

    The level can be then increased to 1.5 grams (1/2 blue scoop) one, two or three times daily thereafter.

    A maintenance level can be 3.0 grams (one blue scoop) two or three times a day necessary for optimum health effects. In stubborn cases this may be increased. The length of time for taking the supplement is an individual response. Supplements need to be taken on a continuing basis for better health--much like vitamins.

    Side Effects: Lauricidin® is a nontoxic nutritional lipid. Lauricidin® may, however, increase the die off of microorganisms and result in a "Herxheimer Reaction," a kind of flu, acne, allergic symptom, etc. You need to cut back on the level or even stop for a few days before taking lower levels and then going higher. Present information on Lauricidin® suggests that it does not interfere with drug metabolism or other supplements. If taken together, Lauricidin® may increase their absorption.*
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    *Statements made here have not been evaluated by the U.S. Food and Drug Administration. These products are not intended to diagnose, treat, cure, or prevent any disease. As with any dietary or nutritional supplement, consult your healthcare practitioner before using natural health products, especially if you are pregnant, nursing, or under medical supervision.

    Disclaimer: Renewal Enterprises does not make claims to consumers in support of health benefits derived from nutritional supplements, vitamins, herbs, diet, or foods. This means we cannot make specific statements as to how nutritional supplements, vitamins, herbs, or diet may help to prevent or treat diseases. Taking natural health products, including recommendations from nutritional consultations, should be a decision based on education and personal research.